oxysporum isolates notably cause disease on many important agronomical crops including grain and pasture legumes (chickpea, Cicer arietinum field pea, Pisum sativum lentil, Lens culinaris lucerne/alfalfa, Medicago sativa), cotton ( Gossypium species), banana ( Musa species) and tomatoe ( Solanum lycopersicum), and was ranked 5th out of the top 10 plant pathogens of scientific/economic importance. truncatula are shared with Arabidopsis in response to F. Our initial results suggest not all components of JA-responses observed in M. truncatula that enable dissection of host jasmonate responses and apply aspects of these herein during the M. We describe publically-available genomic, transcriptomic and genetic (mutant) resources developed in M. oxysporum interactions and compare and contrast this against knowledge from other model pathosystems, in particular Arabidopsis thaliana–F. Here we describe the use of the model legume Medicago truncatula to study legume– F. The root-infecting fungal pathogen Fusarium oxysporum initiates a necrotrophic growth phase towards the later stages of its lifecycle and is responsible for devastating Fusarium wilt disease on numerous legume crops worldwide. Jasmonate (JA)-mediated defences play important roles in host responses to pathogen attack, in particular to necrotrophic fungal pathogens that kill host cells in order to extract nutrients and live off the dead plant tissue.
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